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people may be born with a predisposition toward developing hypertension, according
to a study reported in the Jan. 9th issue of the New England Journal of Medicine.
German researchers have confirmed that the presence at birth of a relatively
low number of nephrons (the functional units of the kidney) is a major contributor
to the development of hypertension. The
researchers compared the number and volume of glomeruli, which make up nephrons,
in 10 middle-aged white accident victims with a history of primary hypertension
with a like number of accident victims with normal blood pressure. Patients
with hypertension had fewer glomeruli, a larger glomerular volume and more severe
arteriolosclerosis than did the controls. The low volume may gradually damage
the kidney as a result of the increased workload per nephron. Lead
researcher Dr. Kerstin Amann, of the University of Erlangen-Nurnberg in Germany,
said it is difficult to know just how the findings will one day impact the treatment
of hypertension. "Hypertension
is certainly a heterogeneous disease," Amann told Medical Week. "We
could show that in some patients the kidney and in particular a low nephron number
is responsible, but in other patients, a gene polymorphism is the cause of hypertension." However,
the findings could lead to an earlier diagnosis of hypertension for patients at
risk and to earlier treatment. "As far as the kidney is concerned several
agents in particular ACE-inhibitors are known to be extremely nephroprotective,"
said Amann. "Thus, for some patients earlier diagnosis can definitely improve
the prognosis." Other
sources: New England Journal of Medicine (Volume 348:101-108)
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